References for Bigelow Tea Health Brochure

References for Health Brochure by Bigelow Tea



Venables MC, Hulston CJ, Cox HR, Jeukendrup AE. Green tea extract ingestion, fat oxidation, and glucose tolerance in healthy humans. Am J Clin Nutr. 2008 Mar; 87(3):778-84.




Steptoe, A, Gibson EL, Vuononvirta R, Willaims ED, Hamer M, Rycroft JA, Erusalimsky JD, Wardle J. The effects of tea on psychophysiological stress responsivity and post-stress recovery: a randomized double-blind trial. Psychopharmacology (Berl) 2007 Jan; 190(1):81-9.


Mukamal KJ, Maclure M, Muller JE, Sherwood JB, Mittleman MA. Tea
consumption and mortality after acute myocardial infarction. Circulation
2002; 105(21):2476-81.


Sesso HD, Gaziano JM, Buring JE, Hennekens CH. Coffee and tea intake
and the risk of myocardial infarction. Am J Epidemiol. 1999; 149(2):162-7.



Kimura K, Ozeki M, Juneja LR, Ohira H. l-Theanine reduces psychological and physiological stress responses. Biol Psychol. 2007 Jan; 74(1):39-45.


Nobre AC, Rao A, Owen GN. L-theanine, a natural constituent in tea, and its effect on mental state. Asia Pac J Clin Nutr. 2008;17 Suppl 1:167-8.


Rogers PJ, Smith JE, Heatherley SV, Pleydell-Pearce CW. Time for tea: mood, blood pressure and cognitive performance effects of caffeine and theanine administered alone and together. Psychopharmacology (Berl). 2008 Jan; 195(4):569-77.


Haskell CF, Kennedy DO, Milne AL, Wesnes KA, Scholey AB. The effect of L-theanine, caffeine and their combination on cognition and mood. Biol Psychol. 2008 Feb; 77(2):113-22.


Kamath AB, Wang L, Das H, Li L, Reinhold VN, Bukowski JF. Antigens in tea-beverage prime human Vy2V82 T cells in vitro and in vivo for memory and nonmemory antibacterial cytokine responses. PNAS. 2003 May; 100(10):6009-14.


Bone Density


Devine A, Hodgson JM, Dick IM, Price RL. Tea drinking is associated with benefits on bone density in older women. Am J Clin Nutr. 2007 Oct; 86(4):1243-7.


Brain Activit/Alzheimer's


Chou CC, Lin LL, Chung KT. Antimicrobial activity of tea as affected by
the degree of fermentation and manufacturing season. Intl J Food Micro
1999; 48(2):125-30.


 Matsunaga K, Klein TW, Friedman H, Yamamoto Y. Epigallocatechin gallate, a potential immunomodulatory agent of tea components, diminishes cigarette smoke condensate-induced suppression of anti-Legionella pneumophila activity and cytokine responses of alveolar macrophages. Clin Diagn Lab Immunol. 2002; 9(4):864-71.


Sonoda J, Koriyama C, Yamamoto S, Kozako T, Li HC, Lema C, Yashiki S, Fujiyoshi T, Yoshinaga M, Nagata Y, Akiba S, Takezaki T, Yamada K, Sonoda S. HTLV-1 provirus load in peripheral blood lymphocytes of HTLV-1 carriers is diminished by green tea drinking. Cancer Sci. 2004; 95(7):596-601


Yam TS, Hamilton-Miller JMT, Shah, S. The effect of a component of tea
(Camellia sinensis) on methicillin resistance, PBP2' synthesis, and
b-lactamase production in Staphylococcus aureus. Journal of Antimicrobial
Chemotherapy. 1998; 42:211-16.




Mitscher LA, Jung, M, Shankel D, Dou, JH, Steele, L, Pillai, SP.
Chemoprotection: A review of the potential therapeutic antioxidant
properties of green tea (Camellia sinensis) and certain of its components.
Med Res Rev. 1997; 17(4):327-65.

Yang, C.S., Hong, Z-Y. Tea and cancer. Journal of the National Cancer Institute, 1993; 85:1038.



Prior RL, Cao G. Antioxidant capacity and polyphenolic components of
teas: implications for altering in vivo antioxidant status. Proc Soc Exp
Biol Med. 1999; 220(4):255-61.


Burns Calories


Dulloo, AG. Am J Clin Nutr 1999; 70:1040-5.




Tan LC, Koh WP, Yuan JM, Wang R, Au WL, Tan JH, Tan EK, Yu MC. Differential effects of black tea vs green tea on risk of Parkinson's disease in the Singapore Chinese Health Study. Amer J Epid 2008 Mar; 1;167(5):553-60. Reference April 17, 2002


Green tea polyphenols to fight Parkinson's


Related topics: Research

The powerful antioxidant properties of polyphenols in green tea could help protect against the onset of Parkinson's disease, according to new research from Baylor College of Medicine in Houston.

Polyphenols in green tea have already been shown to help protect against cancer and heart disease, and indeed earlier studies had also shown that they might protect against Parkinson's disease. However, this is the first time that the way in which they act to protect against the disease has been shown.

Speaking at the annual meeting of the American Academy of Neurology earlier this week, the study's lead author Tianhong Pan said that Parkinson's disease was characterised by the selective cell death of dopaminergic neurons in the midbrain called substantia nigra and by a marked decrease in dopamine neurotransmitter produced by these neurons.

"We tested the levels of dopamine uptake density in a controlled study of mice and the protective effects on dopaminergic neurons against neurotoxin MPP+-induced injury before and after treatment with polyphenol," said Pan.

"Our results indicate the mechanism, or action, of polyphenol is to inhibit the uptake of dopamine or MPP+ by blocking dopamine transporter (DAT), suggesting that its protective effect in Parkinson's disease is its ability to block the DAT-dependent uptake of environmental neurotoxin."

He stressed that the clinical effects of polyphenols on human Parkinson's disease patients remain to be studied.

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